Phyllis Wise accepted the position of Chief Executive Officer of the Colorado Longitudinal Study in 2017.
She has been a leader in higher education and a biomedical researcher.
When Sarah Gray received the devastating news that her unborn twin son Thomas was diagnosed with anencephaly, a terminal condition that meant he would not survive long beyond birth, she decided she wanted his death—and life—to have meaning.
HIV and Hepatitis B and C (HBV and HCV, respectively) viruses cause significant morbidity and mortality worldwide. HCV and HBV kill more people annually than the next 60 reportable infectious diseases combined. HCV related deaths have set new records in every year since at least 2003
Thirty seven percent of the children under the age of 5 in rural Africa are stunted due to chronic hunger and malnutrition. Improving the nutrition of the fundamental food system of Africa, which has not been studied by science, and the translation to scale are crucial to eradicating stunting.
When it’s not too busy being utterly pointless and depressing, The Universe can be funny; really, really, really funny. Just when everything makes sense, nature throws pi in your face, and it turns out we’ve got it all backwards, inside-out, and need to start over from scratch.
What are the market, policy, and financing trends that will impact how states manage Medicaid in 2018 and beyond? How are states connecting the need for health care coverage with improving the overall health of a community?
Carl R. Woese, of Urbana, Illinois, was the man who invented molecular phylogenetics. Although famous to researchers in molecular evolution and microbiology—revered by many, suspect to some—he remains almost entirely unknown to the general public. Even among organismic biologists, ecologists, and evolutionary biologists who study flora and fauna, the name Woese draws a blank.
Clyde’s team used whole-genome design and complete chemical synthesis to minimize the 1079–kilobase pair synthetic genome of Mycoplasma mycoides JCVI-syn1.0. An initial design, based on collective knowledge of molecular biology combined with limited transposon mutagenesis data, failed to produce a viable cell. Improved transposon mutagenesis methods revealed a class of quasi-essential genes that are needed for robust -growth, explaining the failure of our initial design.
The current interest in sequencing the genomes of cancer cells has distracted attention away from the role of epigenetic programs, which may in the end be responsible for the lion’s share of distinct cancer cell-associated phenotypes.